Saturated with Science
The Truth About Saturated Fat
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Saturated fats and their role in health
Hello, dear peoples from all corners!
Today’s lesson is on a very important substance. So dangerous, that it has been blamed in the deaths of millions. So toxic is this, that it is still warned against by the authorities as a disease-causing, epidemic-inducing poison. It has been implicated in the spread of cardiovascular disease, cancer, obesity, diabetes, high blood pressure, and even alien invasions. This substance is so deadly, our bodies need it!
Okay I’m done being a snot. Now for a more serious tone, but not too serious because I don’t want to sound like the Clear Eyes dude. Ahem. Saturated fatty acids are implicated in heart disease and other diseases, and reports abound that lowering it reduced the cardiovascular disease [CVD] rise. This completely ignores the reduction in smoking with the rise in awareness that it caused all the cardiovascular diseases “In addition, age-adjusted death rates per 100,000 persons (standardized to the 1940 population) for heart disease (i.e., coronary heart disease) have decreased from 307.4 in 1950 to 134.6 in 1996 … . During 1964-1992, approximately 1.6 million deaths caused by smoking were prevented … .” (1). Way to go using this victory to blame saturated fat. Bravo. Further, CVD and other diseases are now at an all-time high, or at least was for a vast majority of people, where saturated fat was at an all-time low (2), which negates this entirely as causal. The association of saturated fat with CVD dates back to Ancel Keys and the Lipid Hypothesis (3), in which dietary cholesterol was first blamed, but because specimens had a neutral response where serum cholesterol was almost unchanged, he set his focus on saturated fats.
Saturated fats do not affect serum cholesterol, and courtesy of Dr. Malcolm Kendrick and biochemical sciences (4), this explains why:
The absorbed saturated fat is then packed into a very large lipoprotein (known as a chylomicron). Once a chylomicron is formed it travels up a special tube, called the thoracic duct, and is released directly into the blood stream. It does not, and this is important, pass through the liver.
Chylomicrons then travel around the body and are stripped of their fat, shrinking down until they become about the size of an LDL. At which point they are called chylomicron remnants. These are absorbed back into the liver – using LDL receptors – and are then broken down into their constituent parts
Therefore, a small amount of fat that you eat will end up in the liver. However, the vast, vast, majority will go straight from the guts to fat cells (adipose tissue). Whereupon they are stored away for later use.
In fact, this is the fate of all types of fat: saturated, polyunsaturated, or monounsaturated. There is nothing unique about saturated fat in the way that it is absorbed and transported around the body. Anyway, as you may have noticed, none of this has anything to do with LDL whatsoever. Nothing. Ergo the consumption of saturated fat, or any fat, can have no direct impact on LDL levels.
So there’s the most glaring flaw in this theory, and it was based on flawed data, described in a paper by Denise Minger, an author and opponent of the food guidelines. Her views are unbiased and very detailed in every paper thus far I have read, where she uses the most credible evidence on both sides. She even provides evidence that he did not, in fact, place blame on dietary cholesterol because his controlled trials showed it simply does not happen (5). Another serious flaw is the fact that alone, saturated fatty acids not only do not affect cholesterol, they don’t even have an effect on circulating fatty acids (6).
The evidence against saturated fat is lacking (7-8), and the push for unsaturated fats prompted review because of the swift increase in disease (9-11).
In fact, saturated fatty acids and natural trans fats are quite healthy! CLA is a trans fatty acid found in dairy fat. They protect from cardiovascular disease and events (12), aid in weight loss (13-15), reduce abdominal obesity and have medicinal properties (16-19), among many other benefits. The lungs require saturated fats in the diet, whereas unsaturated fats negatively affect function and even cellular composition (20-22).
SFA intake was inversely associated with mortality from total stroke, including intraparenchymal hemorrhage and ischemic stroke subtypes, in this Japanese cohort.
Inversely associated = prevented or reversed the risk/cured. The ingestion of saturated fatty acids was protective of this often deadly occurrence (12). This is completely contrary to what we’ve been told as a whole and contradictory to the current methods in place.
In fact, this specific kind of disease and others can be attributed to seed foods and their proteins called agglutinins (23-26). This works by a process known as agglutination, which is the binding together or clotting of proteins of a specific type (27). These foods also contain lectins, a type of inflammatory protein. Lectins, in and of themselves, cause problems for a large amount of people, and from specific sources such as grains and legumes [all seeds have them], and for some people eggs [the white], or dairy, (28-32).
Other diseases plunked onto saturated fat? Well, those can be explained too, like PUFA or polyunsaturated fatty acids (33-36); sugar in cancer (37-40)—also involved in a cover-up when the sugar industry found their product to cause cardiovascular disease and cancer (41)—and other diseases (42-45); excessive general carbohydrate (46-53); alcohol (54-55); and many other things which had been postulated as healthful.
The link between animal products [meat and saturated fat pretty much go hand-in-hand] and disease is nonexistent (56-58), with studies even retracting previous associations after correcting for variables (59).
Processed “meat” ingredients: hot dogs
Mechanically Separated Chicken, Water, Pork, Dextrose, Modified Corn Starch, Salt, Contains 2% or Less of the Following: Beef, Corn Syrup, Flavorings, Sodium Phosphates, Potassium Lactate, Potassium Acetate, Sodium Diacetate, Sodium Erythorbate, Oleoresin of Paprika, Sodium Nitrite, Smoke Flavoring.
So, some chicken, water, pork, sugar, sugar, salt, beef, sugar, petroleum by-products, random isolated chemical names that may or may not be natural, a weird mix of paprika instead of just using, you know…paprika, and some random concoction instead of actually smoking the meat.
All these random and scientific jargon ingredients and they said it was the meat?
Say now, fair spirit, that you jest poor little Puck?
You see, this is why all claims against saturated fat can be dismissed as junk science, pseudoscience, and even as propaganda or fearmongering. That and the fact that we evolved and thrived on it since the dawn of human beings, and only recently has the health crisis happened, and continued to worsen the more the misinformation is heeded as legitimate.
I have more studies but I scrolled up and realized this is already long…
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2. Credit Suisse. YouTube video. 18 Jan 2016. The Truth on Fat. Retrieved from
The Lipid Hypothesis
3. “lipid hypothesis.” McGraw-Hill Concise Dictionary of Modern Medicine. 2002. The McGraw-Hill Companies, Inc. Accessed 1 Sep. 2018 from
4. Malcolm Kendrick, MD. Why saturated fat cannot raise cholesterol levels (LDL levels). Accessed 3rd July 2018 from
5. Denise Minger. (22 Dec 2011) THE TRUTH ABOUT ANCEL KEYS: WE’VE ALL GOT IT WRONG. Retrieved on 2 Sept 2018 from
Saturated fat evidence
6. Volk BM, et al. (2014) Effects of Step-Wise Increases in Dietary Carbohydrate on Circulating Saturated Fatty Acids and Palmitoleic Acid in Adults with Metabolic Syndrome. PLoS ONE 9(11): e113605.
7. Patty W Siri-Tarino, et al. Meta-analysis of prospective cohort studies evaluating the association of saturated fat with cardiovascular disease. The American Journal of Clinical Nutrition, Volume 91, Issue 3, 1 March 2010, Pages 535–546,
8 . de Souza Russell J, Mente Andrew, Maroleanu Adriana, Cozma Adrian I, Ha Vanessa, Kishibe Teruko et al. Intake of saturated and trans unsaturated fatty acids and risk of all cause mortality, cardiovascular disease, and type 2 diabetes: systematic review and meta-analysis of observational studies BMJ 2015; 351 :h3978
9. Hamley S. The effect of replacing saturated fat with mostly n-6 polyunsaturated fat on coronary heart disease: a meta-analysis of randomised controlled trials. Nutr J. 2017 May 19;16(1):30. doi: 10.1186/s12937-017-0254-5.
10. James J DiNicolantonio. The cardiometabolic consequences of replacing saturated fats with carbohydrates or Ω-6 polyunsaturated fats: Do the dietary guidelines have it wrong? Open Heart. 8 Feb 2014; 1(1): e000032; doi: 10.1136/openhrt-2013-000032
11. Uffe Ravnskov, MD, PhD; James J. DiNicolantonio, PharmD; Zoë Harcombe, MA; Fred A. Kummerow, PhD; Harumi Okuyama, MD, PhD; and Nicolai Worm, MD, PhD. The Questionable Benefits of Exchanging Saturated Fat With Polyunsaturated Fat. Mayo Clin Proc. April 2014;89(4):451-453;
Saturated fat benefits
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19. Fabian M. Dayrit. The Properties of Lauric Acid and Their Significance in Coconut Oil. Journal of the American Oil Chemists' Society. January 2015, Volume 92, Issue 1, pp 1–15
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21. Marla Paul. (20 May 2014.) Vitamin E in Canola and Other Oils Hurts Lungs. Accessed 2 Sept 2018 from
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Lectins and agglutinins
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25. Pusztai A, et al. Antinutritive effects of wheat-germ agglutinin and other N-acetylglucosamine-specific lectins. Br J Nutr. 1993 Jul;70(1):313-21.
26. Zhao Q, et al. Peanut agglutinin appearance in the blood circulation after peanut ingestion mimics the action of endogenous galectin-3 to promote metastasis by interaction with cancer-associated MUC1. Carcinogenesis. 2014 Dec;35(12):2815-21. doi: 10.1093/carcin/bgu216.
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34. Helmut Bartsch, et al. Dietary polyunsaturated fatty acids and cancers of the breast and colorectum: emerging evidence for their role as risk modifiers. Carcinogenesis, Volume 20, Issue 12, 1 December 1999, Pages 2209–2218,
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Meat and disease
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